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Perfusion
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Reoperation for a type I aortic dissection: case report

Robert K Wise

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Ned T Wiggs

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Marcelo C Dasilva

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Robert M McCoach

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Walter E Pae

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Larry D Baer

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Lindy J Hamilton

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Gregory J Hummer

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Christopher J Kapp

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Dennis R Williams

The Milton S Hershey Medical Center, Penn State Geisinger Health System, Hershey, Pennsylvannia

Surgery for the repair of a type I aortic dissection presents several difficulties for the surgeon and the perfusionist. One must safely support the patient, while at the same time provide the surgeon with a bloodless field in which to operate. Often, this requires cessation of the circulation for varying amounts of time. Deep hypothermia allows for an extension of the arrest period, while other techniques - retrograde cerebral perfusion and antegrade cerebral perfusion - provide an additional degree of cerebral protection.

Recently, we utilized these techniques concurrently on a 43-year-old female who presented for a reoperation for a type I aortic dissection. Combining these techniques allowed us to adequately support the patient during an anticipated lengthy period of circulatory arrest and insured a successful operation without any adverse cerebral or other organ dysfunction.

Perfusion, Vol. 15, No. 2, 155-159 (2000)
DOI: 10.1177/026765910001500211
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